The Shifting Paradigm

I’ve been informed that the management at the WPI has decided that there will be no group in the clinic. The physicians will practice independently, rather than as the academic department I had envisioned. Thus there is no need for a clinical director and I have been given my pink slip, returning to volunteer status. I am disappointed by this decision, but remain completely supportive of the institute’s goals and the scientific effort. I still hope that further consideration will be given to my ideas in the future, when there are fewer obstacles than now, believing that a group would be a better way to manifest the translational research goals of the institute. I’ve launched the Physician Working Group and hope that the sharing of ideas in an ongoing way by this international group of medical practitioners will bear fruit.

I am relieved that there is no longer a question of who I am speaking for here. I write from my multiple perspectives as a doctor, a patient, the mother of a patient, the wife of a nearly recovered but symptomatic patient and the mother of a healthy son with subclinical signs of the illness. My practice is growing and my intention is to share my clinical impressions going forward. This is the 97th blog I’ve written in 15 months. I’ve never gone back and reread it, but from where I stand now, other than changing the URL to the plural, treatingxmrvs, I still believe pretty much what I’ve written. I am continually refining, but the basic concept hasn’t changed. Our illness is of retroviral origin, plus genetics, environment, injury. But a paradigm shift will be required to understand what has happened. That’s why the etiologic agent has been so hard to find. It isn’t one virus, one illness as required by the old paradigm. There’s too much sequence diversity, as Judy Mikovits and Frank Ruscetti have been reporting since the first negative studies. Too many viruses and parts of viruses. And people are working on it. A group in Wisconsin just uploaded sequences to GenBank that suggest more diversity than everybody has been looking for in all the negative PCR studies: Partial molecular cloning with novel consensus PCR primers of the murine JHK retrovirus of human origin, a variant of the Xenotropic murine leukemia virus-relatedvirus (XMRV): Xenotropic MuLV-related virus 5′ LTR, partial sequence; and gag protein (gag) gene, partial cps

The current technology is knocking at the door of solving it. Deep sequencing is beginning to be available, but still very expensive. XMRV has not yet been fully sequenced. VP62 was made in a lab from pieces of viruses taken from multiple tumors. Does it even occur naturally? It has been shown to be infectious in monkeys, but their antibodies don’t detect the strains present in humans, suggesting it’s different than whatever is infecting those humans. Zhang et al’s findings indicate that normal correct lab technique for retroviruses isn’t good enough for XMRV’s, which are not fastidious like HIV. Normal technique does not prevent spread throughout a clean lab in a matter of days. Therefore the contamination issues are serious and our best hope is that somebody develop a reliable serology test that picks up the whole group. A sensitive enough RT (reverse transcriptase) assay seems like it should work too.

Oddly, just as I was writing this, the following came through on Co-Cure:

The FDA is investigating vaccines for contamination by a variety of latent viruses, including XMRV.  They believe that cancer- and tumor-causing latent viruses may become active during the vaccine manufacturing process, and that some of these viruses are hard to detect with standard methods. 

“Xenotropic murine leukemia virus-related virus (XMRV) is a recently discovered human retrovirus that has been found in both chronic fatigue syndrome and prostate cancer patients. Although these findings need further confirmation, there is a potential safety concern regarding XMRV in cell substrates used in vaccines and in transmission by blood transfusion and blood products. We are developing sensitive detection assays for XMRV to evaluate cell substrates and investigate virus transmission by blood transfusion in a monkey model.”

Read the entire article at

This is truly wonderful news!

From Age of Autism and Kent Heckenlively:
FDA to Investigate Vaccines for XMRV Retrovirus

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37 thoughts on “The Shifting Paradigm

  1. >I'm truly sorry to hear of your news- but I know your invaluable contribution and collaberation will continue. I appreciate your integrity, as always.

    BTW- Be sure to keep a screen shot of that FDA page- those kinds of things tend to change and/or get lost. Coming from the autism community, I've seen lots of pages "get lost".


  2. >Thank you for spearheading the sharing of information among physicians, internationally. This is so important to us all, and I'm grateful that you recognize it and are so generously putting your energy into making it happen.

  3. >Thank you, Dr. Deckoff-Jones for continuing to share your experiences, giving hope to all of us. I appreciate your professionalism as well as your kindness.

    Hopefully, this decision will release you from administrative tasks and will allow you to focus on what I assume is your real goal: helping sick people get well. Now you can devote your energies to your family, your practice, your patients, and, I hope, your blog readers. The information you are sharing here is changing lives, and I hope you are aware of how much it is appreciated.

    Patricia Carter

  4. >Thank you, Dr. Deckoff-Jones! You are a life line for so many of us.

    Fascinating recognition from the FDA … thank you for sharing it on your blog.


  5. >Indeed thank you for this blog and everything else, Jamie.

    Is the upshot of this that physicians will have their own software, staff, billing, methodology, etc.?

    Trying to get a more detailed sense.

    In any case I support you and the WPI without reservation as always.

    I hope we will keep hearing from you on this blog.

  6. >Hey Doc,

    bigthank you for all you have done for all of us on both sides of the water.

    I look forward to your coming blog posts.


  7. >Thanks Jamie for all you do.

    Not sure I understand all the politics going on. Thank you for your continual blogging.

    I can't tell you how much I appreciate it.

    ~ JT

  8. >Dear Dr. Jamie Deckoff-Jones! For 2 month ago I read your blog and comments from the beginning, in the spring 2010. I have learned a lot and I have got a little help to treatment. But first og all I will thank you that I have beeing informed over så much.
    Uninformation gives frustration and stress, information in this field give me hope, peace and quiet -it is good for my sickness.
    We are many ME-seek around the world, who never have meet a ME-specialist and for me and others, your blog is maybe is the best source we have. You insight and knowledge fill a vacuum.
    I am thankfull for all you given me and are very thankfull if you have time and strength to continue. It is not a matter of course, it is a gift to us!

    Excuse my bad english.

    Many greetings to you and to you all, and specielt greetings to you who are severe seek. I know a lot how it is.

    Christina Pedersen, Denmark

  9. >Greetings,
    I can't tell you how much reading your blog means to me. It has been a lantern. Thank you.

  10. >Oh boy, looks like we will all need to travel to Hawaii. What a shame! I tend to agree with you (You already knew that). I think the clinic in Reno would probably have a better game plan if they work together and select treatments as a group. This would even facilitate better hypotheses as to what is causal and what works for treatment in CFS.

    Just my two million dollars worth of opinion.

    Paula with NPD

  11. >Dearest Jamie,
    I am saddened by the news, and it is probably for FINANCIAL rather than philosophical reasons, but that is just a guess. I have carried around a poster for 22 years saying "different insult, same result". and i have another poster that is from Louis Pasteur (no dummny!) who said, "THE ANTIGEN IS NOTHING! the TERRAIN is EVERYTHING!"
    and Dr. Paul Cheney has claimed for twenty years that " a healthy person, is like a submarine with a metal door, with a triple lock rubber seal!" . The ME/CFS person has the same submarine, but a SCREEN door! they can float along the surface ok in calm seas, but if they submerge or hit rough seas, they are SUNK, because every kind of insult, be it viral, bacterial or chemical will SWAMP them" (this is paraphrasing i don't evern word correct, but he did use the submarine analogy with the two different doors to explain his theory! i am NOT a doctor, but i play one on TV on Shows like CNN's Larry king live (TWICE) where i compared gulf war illnesses to M.E. and the MacNeil/Lehrer news hout. and i have been a consultant for other CNN shows, and 48 hours and Dateline NBC and the L.A. TIMES, the N.Y.. Times and the Wall Street Journal. i am positive for Lyme, babesia, bartonella, erhlichia, HHV6 and i want to try hyperbaric oxygen chambers next to KILL off the tiny bugs in my spine and brainstem! all the best TO YOU dr. JAMIE, you are a brilliant, compassionate, ethical Human being and i sure wish we could CLONE YOU!!! xoxoo TMH

  12. >In 1985, the CDC's "CEBV Syndrome" hypothesis was based on a concept of "EBV plus some stressor": A state of immune depletion that left one vulnerable to viruses.

    "CFS" was coined, for this and other reasons:

    The illness was characterized by being the exact OPPOSITE, a state of constant immune hperactivation.

    Most people I know have not had a cold or flu ever since they became ill with CFS.
    Or if they do, it is only after their TH1/TH2 flips.

    Immune depletion could be from just about anything, but we know of very few things that can cause chronic activation like this.
    THAT's part of what makes CFS different than what most people think it is.

  13. >Thank you Jamie for this from your prior blog post:

    "To answer the questions above about oxygen. Ali and I are using a concentrator that can deliver 10 L/min which allows use of a non-rebreather mask, so high-dose."

    Can you tell me how long your sessions with the O2 are? I want to investigate this some more, and see if I can get a doctor I know to help me procure an O2 concentrator. I have tanks with an O2 regulator, but that only puts out 6 L/min maximum. The O2 helps me alleviate some of my MCS symptoms, but so far I have only used it during toxic reactions, and not on a regular basis.

    Any other info you have or can recommend on high-dose oxygen would be appreciated.

    Thanks again. ~~DB

  14. >Dear Jamie,
    Sorry to hear that the vision you had of your role at the WPI didn't quite pan out as you may have hoped. I was disappointed as I read that bit of information but I was greatly heartened to read about the recent FDA vaccine contamination investigation announcement.
    Every time I read your blog I feel that we are all getting a little closer to knowing the truth behind our illness and it really cheers me along (and oh how I need a bit of cheering up, trying to look after my 3 kids during the 6 week summer holiday here in the UK!! :)
    You continue to bring relevant and interesting information to many of us who don't have the energy or concentration to find it ourselves – I am so thankful for this.

  15. >*********************************
    Just in case Dr. Snyderman has not seen this:

    …"Levine and his colleagues designed a new gene that can be inserted into T cells to trick them into attacking cancerous B cells, the cause of chronic lymphocytic leukemia (CLL). The new gene encodes a receptor that, on one end, can bind to a molecule that's unique to cancerous B cells. The other end of the receptor sets off a chain reaction when such a B cell is bound, eventually leading the T cell to destroy the cancerous cell. "Essentially, we're converting T cells that would normally recognize other types of cells to be tumor specific," Levine says.
    In the initial clinical trial, the researchers tested their method in three patients with CLL. They took a sample of each patient's T cells and added the new gene to the cells, using the same procedure and new gene in each case, while keeping the T cells from each patient separate. Once the T cells contained the new, tumor-specific receptors, the scientists infused the T cells back into the blood of each patient.
    All three patients are now in remission. On average, the modified T cells killed over a kilogram of tumor cells in each patient. For every modified T cell infused into the patients' blood, at least 1000 tumor cells were killed, leading the researchers to dub the T cells "serial killers." Moreover, after 12 months, blood tests revealed that the patients still had copies of the modified T cells circulating in their bloodstream able to kill cancer cells. And although the data spans only a year, Levine says the effects are likely even longer…"

    From Science Mag article:

    Full Study:

    Jerry and Carol
    El Paso

    PS. I think in the long run Dr. Deckoff-Jones (and family) will end up better off with this development, as will patients. Much better to test ideas in many different parts of the world than just there in Reno. With the 'net', we are all connected at light speed anyway…

    Oh, and for the record, two days after receiving my oral polio vaccine back in '60 I was in the hospital getting an IV and being treated for a severe 'flu'…just a coincidence they said…"nothing to see here, move on – move on…". In Feb of 83 in El Paso there was an outbreak of folks getting mono and what is now called hepatitis C. My own doc said he had seen over 60 patients like me, and on my next appointment there was someone from the CDC (Center for Disease Concealment) in the room…"this is Dr So and So from Atlanta". I did not realize it back then though, just through hindsight. But I am the rarest of rare, a cfs patient whose first doc knew I had a real, 'new' disease.

  16. >That's a great story, Jerry.
    I've got one of those "rare" stories too.

    My very first doctor was a really nice guy and believed me straight off. I never saw a shred of doubt from him that my illness was real.
    He ordered brain scans, every test he could think of, and tried really hard to help me.
    But he finally said "We've reached an impasse.
    I've never seen anything like this and don't know where to go from here. I've run out of tests. It would probably be best for you to try going to the large clinics, who might be able to recognize your illness"… and dismissed me.
    Regretfully, I left his practice and went to the big clinics, and ran through ten doctors who all told me "IAIYH".
    Then one day I saw a story in the local newspaper that my first doctor and his partner had identified a few dozen cases of a mysterious fatiguing illness in our little town.
    I showed it to the doctor I was seeing, and he said, "Yes, I know about that, but those two doctors are quacks. They should be tarred and feathered, and run out of town on a rail".
    Pointing at the paper, I retorted, "But THESE are all my symptoms, I have the same thing".
    Unable to contain myself any further, I yelled at him, "YOU KNEW??? But WEREN'T GOING TO TELL ME???" I was furious.
    I immediately walked out of this guys office, closing the door "with vigor", and went straight back to my very first doctor, the one who had believed me… Dr Paul Cheney, who welcomed me back.

  17. >Dr. Cheney understands the pathophysiology of this illness better than any other doctor out there. He's also the most brilliant, holding a PhD in atomic physics, which has enabled him to think outside the box. And he doesn't discount mold or any other number of "co-factors," as he is treating TERRAIN and redox buffering capacity. But I think he believes that a retrovirus is the common denominator. So do I.

    And while I have all of the hallmarks of this disease in terms of Gerwyn's cytokine profile, low NK counts,RNAseL irregularities, chronic inflammation, mycoplasma infection, elevated Herpes viral titers, disturbed sleep, etc, there was a time early in my illness when I was severely immune depleted and had Aids-related complex after two years of infection without HIV (ARC generally appears after a decade). Also, my experience echoes Dr. Decker-Jones in that I could still exercise and do other things, despite significant malaise, feeling "crushed" in the morning, and brain inertia. Then it all went downhill. My sister has this, my brother has a "fatiguing illness," my wife became infected, though she recovered in the same environment I was living in, and my dad had an idiopathic meningitis that almost took his life and paralyzed one of his eyes. He developed colon cancer some ten years later. We all developed the illness in different time frames with exposure to different environments. My wife certainly had no commonality.

    As Jamie blogged early on, the virus has a gluccorticoid promoter region, genetics are likely involved in addition to environment, and vertical transmission is obvious. As they say, "It's the virus, stupid."

  18. >Dr Cheney and the entire medical profession ARE discounting the mold… STILL.
    They show no signs of looking into it and are not giving patients advice that would be considered commensurate with taking it seriously.

    After I agreed to serve as a prototype for "a new syndrome", I left Dr Cheney/Peterson's practice BEFORE "CFS" was created.
    People couldn't understand why I would do this, when they were at the absolute lead in researching this illness entity.

    Several others of the Incline group and I were getting better results by "avoidance" than anything that various therapies were accomplishing, so this is what we did.
    To this day, there has been no investigation into this aspect of the phenomenon.
    To me, that is discounting it.

  19. >To Anon at 8/11, 1:27 PM:

    I have a great deal of respect for Paul Cheney and am very glad that he has chosen to devote his life to the study of this illness.

    I also was glad to hear that, about a year ago, he wrote an email to his patients urging them to look for toxic mold in their homes and to address the situation (preferably by moving) if they found it.

    I was informed that a subsequent study of his patients last fall showed that those who knew that they had had significant mold exposure were substantially more likely to come up positive for XMRV than those who had not had exposure (or were unaware of having been exposed).

    This is consistent with what we know about XMRV — that it spreads as a result of inflammation such as is generated by exposures to toxic mold. An article summary (which I’m sure you’re familiar with) is below.

    If we can’t yet effectively address the retrovirus directly with drugs, addressing injury and environment to inhibit its spread seems to be our best option for dealing with it. Cheney’s concept of addressing the “terrain” thus makes sense to me.

    It’s been a while since I’ve heard of him bringing up mold either to his patients or in public though.

    Do you know if he’s still counseling people to take active steps to avoid it or doing any more research into the topic?

    Thanks much for your comments.



    lisapetrison at yahoo


    Sakakibara S, Sakakibara K, Tosato G. NF-kappaB activation stimulates transcription and replication of retrovirus XMRV in human B-lineage and prostate carcinoma cells. J Virol. 2011 Apr;85(7):3179-86. PMID: 21270144

    Xenotropic murine leukemia virus-related virus (XMRV) is a gammaretrovirus linked to prostate carcinoma and chronic fatigue syndrome. Here we report that NF-κB activation can markedly increase XMRV production. The inflammatory cytokine tumor necrosis factor alpha (TNF-α), which activates NF-κB, significantly augmented viral Gag protein production in XMRV-infected cells. Reporter assays showed that TNF-α and Epstein-Barr virus (EBV) latent membrane protein 1 (LMP1), an intrinsic NF-κB activator, increased long terminal repeat (LTR)-dependent XMRV transcription. We identified two NF-κB binding sites (designated κB-1 and κB-2) in the LTR U3 region of XMRV and demonstrated that both sites bind to the NF-κB component p65/RelA. Mutation of the κB-1 site, but not the κB-2 site, impaired responsiveness to TNF-α and LMP1 in reporter assays. A mutant XMRV with a mutation at the κB-1 site replicated significantly less efficiently than the wild-type XMRV in the prostate carcinoma LNCaP, DU145, and PC-3 cell lines, HEK293 cells, the EBV-immortalized cell line IB4, and the Burkitt's lymphoma cell line BJAB. These results demonstrate that TNF-α and EBV LMP1 enhance XMRV replication in prostate carcinoma and B-lineage cells through the κB-1 site in the XMRV LTR, suggesting that inflammation, EBV infection, and other conditions leading to NF-κB activation may promote XMRV spread in humans.

  20. ><<
    Jerry and Carol
    El Paso>>

    Jerry, I'm in El Paso as well and would like to talk to you. Would you please contact me?


  21. >Dr. Deckoff Jones, your assessment once again contains incorrect and misleading information. Specifically,

    1. Contrary to your post, there is actually almost no 'sequence diversity' in XMRV. The sequences of XMRV uploaded so far are all almost identical. Instead what has occured is that any virus of the MLV class is being mistakenly called 'XMRV', whether it be an murine endogenous retroviral sequence or a different and distinct recombinant cell line contaminant, which is not accurate. XMRV is a very specific virus and the other sequences uploaded so far represent other distinct viruses and/or murine ERV sequences, not sequence diversity in XMRV.

    2. XMRV has indeed been fully sequenced, XMRV was first fully sequenced several years ago and has been fully sequenced several times since then. As stated above, all of these sequences are nearly identical.

    3. Your point about XMRV antibodies in monkeys is not clear. You state that '(XMRV) has been shown to be infectious in monkeys, but their antibodies don't detect the strains present in humans, suggesting it's different than whatever is infecting those humans'. However the only XMRV isolates obtained from humans are almost exactly 100% identical to the VP-62 clone which the monkeys were infected with, so the fact that the XMRV-specific antibodies obtained from the monkeys does not react with human samples indicates that the human samples are not in fact infected with XMRV, not that humans are infected with a different strain. The antibody tests done by Lombardi and reportedly Lo as well might possibly indicate something like what you suggest, but the Emory monkey data you present does not support your assertion and in fact actually works against it.

    4. You refer to Zhang et al's findings about MLV retrovirues being spread throughout a lab in a matter of days, and even specifically refer to ease of contamination in this regard, however you do not bring up the obvious issue that this finding is just as relevant to the issue of the WPI's results being contaminated.

    On a plus note, maybe now you'll have more time to catch up on the research you've been telling us all about!

  22. >I'd just like to say that Dr Peterson and Dr Cheney are two of most brilliant and incredibly dedicated physicians I have ever been privileged to start a syndrome with.

    I can well-understand their reaction of disbelief to my bizarre story.

    What I saw, you literally had to be in my hiking boots to believe.

  23. >Lisa, I know that Dr. Cheney has asked people to remediate their homes, and asked one individual to pick up and leave a home that would take well over $100,000 to adequately remediate. He does not discount mold, and I believe he's had conversations with Ritchie (I don't know when Erik last spoke with him), but he also doesn't give it top billing, and I don't believe he should. He refers to bad foods, VOCs, other pathogens, environmental poisons, etc exacerbating the illness, perhaps via activating P450 cytochromes in the liver and kidneys. He also thinks many different co-factors can elevate C4a, not just mycotoxins. There's plenty of evidence to support this.

    My story above is not one consistent with mold, though I don't completely discount it myself. But I think some individuals are MORE susceptible to mold poison than others, making this even more convoluted. I don't believe you can get M.E. without an HGRV present, mold exposure or not.

    The most irritating part of this dialogue to some of us is that, unless you are a single person, married with no kids, or a trust fund baby, taking the draconian steps of "mold avoidance" are all but impossible, even for the most persevering, particularly if you're not on disability. Someone has to pay for this. Think the government, which can ill-afford another pandemic given the artifical nature of this "recovery" propped up by the Fed's funny money, has any interest in doing so?

    Dr. Cheney is the MOST brilliant doctor in the field, bar none.

  24. >Dr J – lisa, anon, and erik not included, your blog has consistently been of immeasurable assistance at several important points in my "treatment", and i use that word loosely, as it allows me to call a few doctors on their bull. thanks and please keep at it – there are many of us to whom you are a light and a doctor worth reading. again – thanks

  25. >and by the way – lisa and erik and whoever anon is that needs to point out the "errors" of your ways – please get your own blogs! a lot of us are sick to death of your hi-jackin ways!

  26. >Dr. Jamie, what's your advice for people who are thinking about mold? Do you agree with Dr. Cheney?

  27. >If this were a popularity contest, the blog administrator for sure would win (amongst those who are still reading her blog).

    Is that what it is, a popularity contest?

    Huh. How weird.

  28. >@Erik,

    I wish I was better at keeping track of things, but I have seen more references lately from docs taking the role of mold seriously. My own naturopath notices a huge increase in fungal-caused illnesses in his own practice.

    You have been in the trenches for a long time, in the front line, and I totally respect that; what I notice in your posts though is a strong attachment to the (understandable) anger about having been not listened to for so long–to the point that you seem to be ignoring and dismissing the very doctors that DO take your point seriously.

    "the entire medical profession ARE discounting the mold… STILL."

    This statement is an extreme POV, and untrue, and I think you yourself might feel better and happier if you take a look at that strongly held belief, and allow a different, evolving (albeit slowly) reality to seep in.

    I am not referring at all here to the role of mold in ME, but to the blinders created by your own "attachment" to the long-held role of being an outsider on this, Relax away from the Outsider a little, and maybe you'll better see the people who actually see what you do. (I see it too, though I am not an MD.)

    OT to this blog, perhaps, but germane to this whole illness and its socio-political-medical evolution: we all have to remain flexible and open to the rapidly developing ideas about NeuroImmune disease, and be careful not to get too locked in to any POV, even our own victimization. The community as a whole must remain open to all possibilities so we can solve this thing.

  29. >Maybe Erik thinks that even those doctors who believe that mold is a factor in CFS don't understand just how bad it can be or what it can do.

    Since the blog administrator has seen fit to delete his comments though, I guess we won't be able to know.

  30. >The blog administrator has deleted nothing that isn't extremely repetitive.

  31. >"Maybe Erik thinks that even those doctors who believe that mold is a factor in CFS don't understand just how bad it can be or what it can do."

    Yes, maybe that is was Erik believes. I don't have a beef with what Erik believes. I just thought it useful to point to the blanket, divisive, extreme expression

    "The Entire Medical Profession "

    This even excludes Erik's friend and collaborator Dr. S.
    I am not picking on Erik at all. I am gently trying to point out how extreme beliefs can sometimes unintentionally limit us in the very thing we are trying to do. This goes for all of us.

  32. >"Dr. Cheney is the MOST brilliant doctor in the field, bar none."

    Then could you please explain why he has the lowest 'success' rate when it comes to patients recovering, let alone even partially improving?

    He's been a strong advocate yes, but as a doctor, his results are abysmal. Let's look at his recent stem-cell 'experiment'. Do you know of even a half dozen patients that have improved and held that improvement? I don't.

    I do know one woman who has traveled south of the border for his unproven treatments — three times — only to get worse and worse. She is now bedridden. This probably cost her about $40-50,000 as well.

    Please explain why he's "the most BRILLIANT".

  33. >How is this getting simpler?

    We started out with the hypothesis there was one virus that was causal of all ME. A very questionable hypothesis, of course, since the constellation of symptoms called ME are multi-causal, multiple pathogens, and toxic insults.

    The virus was said to have a very low mutation rate.

    The HIV literature was cited frequently, even though the parallels seemed questionable (to me). Now HTLV is cited. Now we've gone from causal to, only in the vulnerable.

    Now we have multiple strains apparently from multiple vaccines? I'm not sure. Or mutations? A virus that had a very low mutation rate, now has multiple strains?

    How is this getting simpler?

    Re: mold. If you're sensitized to mold, to the point where it makes you very ill, you are likely going to have a problem in many if not most buildings. Because of the way buildings are built, particularly residential buildings, as well as schools and hospitals. HVACS and the materials they're made of–asking for it. Insulation and the materials they're made of–sitting ducks. Vapor barriers and the materials they're made of–gathering places for wetness. Tight buildings–another win for mold. Bathrooms and kitchens not properly ventilated–go mold! Plywood and pressboard–woo hoo, easy to mold. IMO society can barely take it seriously because it requires too much effort to fix the longstanding problem (from the 70's onward at least…)

    I do agree with the poster who said Cheney thinks lots of assaults make this illness worse–at least for some of us. Others, just seem to have a purer form of whatever this constellation of illnesses is, and respond well to whatever it is they take: the right antibiotic(s), the right antiparasitical drug(s), the right antiviral(s), etc.

    It really doesn't seem to be getting simpler though, to me. The hypothesis seems to be a moving target.

    Sorry I posted as anonymous–this is Jill Neimark, I just have a different google account now.

  34. >Dr Cheney has actually pieced together much of the disease processes. He is an exceptional doctor. But his record in treating the disease is no different to any other successful doctor, because it is not an easy disease to treat. If HGRVs are the cause, as the evidence does indicate, then is it any wonder when the virus integrates into a person genome and is therefore life long.

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